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Muralidhar S

Vitamin D–VDR signaling inhibits Wnt/β-Catenin–mediated melanoma progression and promotes antitumor immunity
Genetic and environmental determinants of immune response to cutaneous melanoma
Primary melanoma expression of the vitamin D receptor (VDR) is protective for melanoma survival and is associated with increased tumor immune response, decreased Wnt/B-catenin signaling and tumor proliferation
β-Catenin–mediated immune evasion pathway frequently operates in primary cutaneous melanomas
Higher serum 25‐hydroxyvitamin D levels at melanoma diagnosis are associated with better prognosis in patients with intermediate VDR‐expressing tumours, with transcriptomic evidence for inhibition of β‐catenin signalling

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